Download e-book for iPad: Age-Related Macular Degeneration by Jennifer I. Lim

By Jennifer I. Lim

ISBN-10: 082470682X

ISBN-13: 9780824706821

ISBN-10: 0824744039

ISBN-13: 9780824744038

This reference showcases exudative and nonexudative AMD therapy innovations utilizing argon and diode lasers, submacular and macular translocation surgical procedure, photodynamic remedy, radiation, feeder vessel therapy, thermotherapy, and antiangiogenesis medicines. together with one hundred sixty colour illustrations, Age-Related Macular Degeneration will profit medical ophthalmologists, pathologists, physiologists, and pharmacologists; optometrists; cellphone and molecular biologists and biochemists; retinal experts; AMD researchers; and graduate and scientific university scholars in those disciplines.

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Extra resources for Age-Related Macular Degeneration

Sample text

Under certain conditions, oxygen-containing molecules can accept an electron from various substrates to become highly reactive products with the potential to damage cellular molecules and inhibit functional properties in pathogens or host cells. Four of the most important oxidants are singlet oxygen, superoxide anion, hydrogen peroxide, and the hydroxyl radical. In addition, various nitrogen oxides, certain metal ions, and other molecules can become reactive oxidants or participate in oxidizing reactions.

Since these disorders share epidemiological, genetic, and physiological associations with AMD, the approach will attempt to delineate the scope of the subject based on analysis of other age-related degenerative diseases, and to highlight areas of potential importance to future AMD research. Finally, this chapter will introduce the paradigm of “response to injury” as a model for AMD pathogenesis. This paradigm proposes that immune mechanisms not only participate in the initiation of injury, but also significantly contribute to abnormal reparative responses resulting in disease pathogenesis and complications.

Antigen trapped in the extracellular matrix). These “in situ” or locally formed complexes sometimes activate the complement pathway to produce complement fragments called anaphylatoxins. This mechanism should be differentiated from deposition of circulating immune complexes, which are preformed in the blood. Typically, the histology is dominated by neutrophils and monocytes, but at low level of activation minimal cellular infiltration may be observed. Many types of glomerulonephritis and vasculitis are thought to represent this mechanism.

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Age-Related Macular Degeneration by Jennifer I. Lim


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